Why Soy?

By Heartspring Staff

Related: Why Not Soy? The side effects.

Soy is a rich source of the isoflavone, genistein and animo acid proteins, researched for their beneficial health properties.

Food and Drug Administration (FDA), U.S. Department of Health and Human Services. 1999. FDA talk Paper: FDA approves Soy Protein and Coronary Heart Disease; T99-48, Oct 20, 1999

Goodman-Gruen D, Kritz-Sliverman D. 2001. Usual dietary isoflavone intake is associated with cardiovascular disease risk factors in postmenopausal women. Journal of Nutr Apr;131(4):1202-6

LE Moeller, CT Peterson, KB Hanson, SB Dent, DL Alekel (SPON: DS Lewis). 2000 Isoflavone-rich soy favorably affects regional fat and lean tissue in menopausal women. Iowa State Univ. Ames 50011. Experimental Biology 2000, San Diego, CA April 15-18

Lee M, Kim I. 2000. Soy protein and obesity. Nutrition Jun;16(6):459-60

Saito M. Effect of soy peptides on energy metabolism in obese animals. Nutrition. Sci. Soy Protein 1991:12:91

Tsai AC, Vinik AI, Lasichak A, Lo GS. 1987 Effects of soy polysaccharide on postprandial plasma glucose, insulin, glucagon, pancreatic polypeptide, somatostatin, and tryglyceride in obese diabetic patients. American Journal of Clinical Nutrition. Mar;45(3):596-601

Sanchez A, Hubbard RW. 1991 Plasma amino acids and the insulin/glucagon ratio as an explanation for dietary protein modulation of atherosclerosis. Med Hypotheses Sep;36(1):27-32

McCarty MF. 2000. The origins of western obesity: a role for animal protein? Medical Hypotheses Mar;54(3):488- 94

Aoyama T. et al. 2000 1986. Soy protein isolate and its hydrolysate reduce body fat of dietary obese rats and genetically obese mice. (yellow KK). Nutrition May;16(5):349-54

Kawano-Takahashi Y. et al. 1986. Effect of soya saponins on gold thioglucose (GTG)-induced obesity in mice. International Journal of Obes 10(4):293-302

Wang GJ, Volkow ND, Logan J, Pappas NR, Wong CT, Zhu W, Netusil N, Fowler JS. 2001. Brain dopamine and obesity. Lancet Feb 3;357(9253):354-7

Bare DJ, Ghetti B, Richter JA. 1995. The tyrosine kinase inhibitor genistein (soy) increases endogenous dopamine release from normal and weaver mutant mouse striatal slices. Journal of Neurochem Nov;65(5):2096-104

Parry-Billings m, Blomstrand E, McAndrew N, Newsholme EA. 1990. A communicational link between skeletal muscle, brains, and cells of the immune system. Int Sports Medicine 2:S122-S128




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